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Many living organisms are able to regenerate damaged or lost tissue, but why some are particularly good at this and others are not is not fully understood. Molecular biologists Alexander Stockinger, Leonie Adelmann and Florian Raible from the Max Perutz Labs at the University of Vienna have now made an important contribution to clarifying this question in a new study.
A study by researchers at the Icahn School of Medicine at Mount Sinai has identified strategies for using large language models (LLMs), a type of artificial intelligence (AI), in health systems while maintaining cost efficiency and performance. The findings, published in the November 18 online issue of npj Digital Medicine, provide insights into how health systems can leverage advanced AI tools to automate tasks efficiently, saving time and reducing operational costs while ensuring these models remain reliable even under high task loads.
Dr. Hye Jung Chang and Dr. Kyung Joong Yoon (Director) of the Hydrogen Energy Materials Research Center at the Korea Institute of Science and Technology (KIST) have announced that they have elucidated the mechanism of the initial degradation phenomenon that triggers the performance drop of high-temperature solid oxide electrolysis cell systems, using advanced transmission electron microscopy.
A study led by UC Davis distinguished professor Andreas Bäumler uncovered how Salmonella, a major cause of food poisoning, can invade the gut despite the presence of protective bacteria.
How ecDNA Fuels Cancer by Breaking the Laws of Biology
Creating a spatial map of the sea
The study findings point to the potential of interleukin-13 antibodies — approved for use in humans — as a treatment to protect against bacterial pneumonia in patients with zinc deficiency.
The rod-shaped tuberculosis (TB) bacterium, which the World Health Organization has once again ranked as the top infectious disease killer globally, is the first single-celled organism ever observed to maintain a consistent growth rate throughout its life cycle. These findings, reported by researchers on November 15 in the journal Nature Microbiology, overturn core beliefs of bacterial cell biology and hint at why the deadly pathogen so readily outmaneuvers our immune system and antibiotics.
SLAC’s SSRL helps pin down key players in the microbial production of methylmercury, a poison that can accumulate in fish.
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