Model of MYC2 in the modulation of fatty acid and flavonoid biosynthesis under MeJA treatment. In the early stage of olive fruit development, accumulation of MeJA could decrease the expression of MYC2, which could repress fatty acid biosynthesis by downregulating the expression of BCCP2 though binding to the G-box within the promoter. Conversely, MYC2 was proved to directly bind to and activate the promoter of FLS, ultimately leading to an increase in flavonoid content.